Seracare軍團菌嗜肺軍團菌陽性對照

廣州健侖生物科技有限公司

廣州健侖長期供應各種生物原料，主要代理品牌：美國Seracare、西班牙Certest、美國Fuller等等。

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Seracare軍團菌嗜肺軍團菌陽性對照

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與此同時，該研究的結果也可以應用于人類機體其他組織，比如皮膚、肺部和抗原抗體等;揭示干細胞在人類腸道中的行為機制將是干細胞研究領域的一大飛躍，截至目前研究人員更多的是利用小鼠來進行研究或者是將干細胞置于自然環境中進行研究，這就扭曲了干細胞的正常行為表現;而這項研究中研究者利用開發的新型工具來揭示腸道干細胞的行為表現，這就可以明顯增加研究人員對于腸癌發生的理解。
zui后，研究者Marnix Jansen博士說道，基于本文的研究，通過對來自腸癌患者的組織進行分析研究，我們就可以清楚揭示腸癌的發生機理，當然后期還需要進行更為深入的研究才能夠為開發治療腸癌患者的疾病以及改善其生存質量提供幫助。
盡管療法已經得到了極大改善，但是兩個成年急性髓性白血病(AML)患者中也僅有一人會生存下去，對于65歲以上的老年人而言，急性髓性白血病平均存活期不到一年;而研究者推測發病原因可能是白血病干細胞，在患者機體中白血病干細胞并不能*被清除，這些白血病干細胞就是引發患者病情復發zui終致死的原因。
近日，刊登在雜志 Cancer Research 上的一篇研究論文中，來自歌德大學( Goethe-Universitat Frankfurt am Main )的研究人員通過研究發現了急性髓性白血病干細胞的弱點，他們發現了一種名為5-脂氧合酶(5-LO)的酶類在急性髓性白血病干細胞的生存過程中扮演著重要角色。
此前研究人員已經發現了 5-LO 在炎性疾病比如哮喘癥發病中的角色，研究者 Jessica Roos 教授表示，這項研究中我們發現，急性髓性白血病亞群中的白血病干細胞或許可以被5-LO抑制劑有效地選擇性攻擊，而且這種現象均可以在細胞培養基模型和白血病小鼠模型中觀察到。
研究者表示，這項研究為開發新型的5-脂氧合酶抑制劑來抑制白血病的存貨，從而為抑制并治療急性髓性白血病提供了新的研究依據和線索。后期研究中研究人員還將通過深入研究來揭示5-脂氧合酶抑制劑在抑制白血病干細胞功能中的作用和機制。
科學家們早就知道，當細胞反復分裂時，就會發生染色體缺陷。隨著時間的推移，這些缺陷就會導致癌癥的發作。
現在，來自英國卡迪夫大學的鄧肯·貝爾德教授和他的研究團隊與來自明尼蘇達大學的埃里克A·亨德里克森合作已經確定了，人類細胞為了生存所需的這些類型的缺陷的一個特定基因。
“我們發現，似乎是在進化過程中，可以驅動癌癥的關鍵基因，” 卡迪夫大學的癌癥與基因研究所的貝爾德教授說。“這是該基因的新作用，使其成為潛在的治療靶點。”
隨著細胞的分裂，它們的端粒——保護染色體末端免受損壞的DNA“帽”——縮短，使殘余的染色體容易彼此粘在一起。
在正常細胞中，這種染色體的粘性是一個死亡的信號，該信號能夠觸發缺陷的細胞在清理過程中移動，并幫助清理它們的過程。
然而，惡性細胞，在某種程度上能夠躲避這個清理過程。

我司還提供其它進口或國產試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、食品安全、化妝品檢測、藥物濫用檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。

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【公司名稱】 廣州健侖生物科技有限公司
【市場部】    楊永漢

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【騰訊  】 2042552662
【公司地址】 廣州清華科技園創新基地番禺石樓鎮創啟路63號二期2幢101-103室

At the same time, the results of this study can be applied to the human body's other tissues, such as skin, lungs and antigen antibodies; reveal the mechanisms of stem cell behavior in the human gut is the field of stem cell research will be a great leap forward, as of now researchers More is the use of mice to carry out research or put the stem cells in the natural environment for research, which distorted the normal behavior of stem cells; and researchers in this study using the development of new tools to reveal the intestinal stem cells Behavioral performance, which can significantly increase the researchers understanding of the occurrence of colorectal cancer.
Finally, the researcher Dr. Marnix Jansen says, research paper based on tissue from patients with colorectal cancer through the analysis, we can clearly reveal the mechanism of cancer, of course, the latter also need more in-depth study to be able to Development and treatment of diseases of patients with colorectal cancer and improve their quality of life to provide help.
Despite the dramatic improvement in therapies, only one in two adults with acute myeloid leukemia (AML) survives, with an average survival of less than one for acute myeloid leukemia in older adults over 65 years Year; and the researchers speculated that the pathogenesis may be leukemia stem cells, leukemia stem cells in the patient's body can not be compley removed, these leukemia stem cells is caused by the patient's condition eventually lethal cause of death.
Recently, in a research paper published in the International Cancer Research, researchers from Goethe-Universitat Frankfurt am Main discovered the weakness of acute myeloid leukemia stem cells through research that they found in a study entitled 5 - Lipoxygenase (5-LO) enzymes play an important role in the survival of acute myelogenous leukemia stem cells.
Previously researchers have found that 5-LO role in the pathogenesis of inflammatory diseases such as asthma in, the researchers Jessica Roos Professor said the study, we found that acute myeloid leukemia subgroup of leukemic stem cells might be 5- LO inhibitors effectively selectively attack, and this phenomenon can be observed in both cell culture and leukemia mouse models.
The researchers said the study provides new research evidence and clues for the suppression and treatment of acute myeloid leukemia in order to develop new 5-lipoxygenase inhibitors to inhibit the stock of leukemia. In the latter part of the study, researchers will further study to reveal the role and mechanism of 5-lipoxygenase inhibitors in inhibiting leukemia stem cell function.
Scientists have long known that chromosomal defects occur when cells repeatedly divide. Over time, these shortcomings can lead to the onset of cancer.
A now from Cardiff University's Professor Duncan Baird and his research team A · Eric Hendrickson from the cooperation with the University of Minnesota have identified, in order to survive in human cells required for these types of defects Specific gene.
"We found it to be a key gene that can drive cancer in the evolutionary process," said Professor Baird, a professor of cancer and genetics at Cardiff University. "This is a new role for the gene, making it a potential therapeutic target."
As the cells divide, their omeres - protecting the chromosome ends from damaged DNA "caps" - shorten the residual chromosomes to stick together easily.
In normal cells, the stickiness of this chromosome is a signal of death that triggers defective cells to move during the cleaning process and help cleanse them.
However, malignant cells, to some extent, are able to evade this cleanup process.