違禁品十一聯快速檢測試劑盒

廣州健侖生物科技有限公司

廣州健侖長期供應各種藥篩檢測試紙、違禁藥物檢測卡、違禁藥品檢測試劑盒、藥篩試紙、藥篩試劑盒等，包括進口和國產的不同品牌。

主營品牌：美國US、美國Alfa、美國NovaBios、美國Cortez、國產創侖等等。

主要用途：篩查違禁品濫用殘留、麻醉類藥物殘留、興奮類藥物殘留等等。

檢測范圍：嗎啡、巴比妥、尼古丁、KET、mamp、MDMA、BZO、THC、MTD、BAR、MDMA、AMP、BUP、PCP、TCA、OXY、MET等等。

產品特點：可以根據需求自主訂制多聯卡。可以自由組合，從二聯到十五聯都可以訂制。

我司還提供其它進口或國產試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、化妝品檢測、食品安全檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。

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美國Alfa 藥篩試劑盒 藥篩試劑盒

尿液試紙、唾液試紙、尼古丁檢測卡、煙堿檢測卡、違違禁品三聯檢測卡、違禁品五聯檢測卡、違禁品十聯檢測卡、藥篩試劑、違禁品濫用檢測試紙、違禁品快速檢測試劑盒

美國Alfa多聯檢測杯簡介：

美國Alfa單卡產品簡介：

【功能介紹】

可以檢測尿液中是否含嗎啡成分。從而定性判斷被測者是否吸食了嗎啡。 

【樣品要求】

用一次性尿杯收集尿樣，無需處理可直接檢測。

【檢驗方法】

1、測試前先閱讀使用說明書；

2、用干凈尿杯取尿樣；

3、從鋁箔袋中取出檢測卡，置于干凈平坦的臺面上，用吸管；垂直滴加2-3滴尿樣到加樣孔中；

4、3-5分鐘讀結果。為確保結果的準確性，請勿在5分鐘后判讀結果。

【結果解釋】

1、陽性：在反應區內只出現一條紅色質控線。

2、陰性：在反應區內出現質控線和反應線兩條紅線。

3、無效：在反應區內質控線未出現，需重新測試。

【注意事項】

1、檢測卡在室溫下一次性使用，不得重復使用；

2、檢測卡從鋁箔袋中取出后應在30分鐘內盡快使用

3、3~5分鐘內判定結果，10分鐘后的結果無效

4、謹防檢測卡受潮，檢測卡受潮或鋁箔袋破損后，檢測卡不能使用

5、由于標本采集時存在差異，檢測過程中可能出現質控線C和反應線T的顏色深淺或明暗不等，但只要可見，不管其顏色深淺或明暗均應視為出現。

違禁品十一聯快速檢測試劑盒

一、動脈性充細菌

動脈性充細菌（arterial hyperemia）又稱主動性充細菌（active hyperemia），簡稱充細菌。一般指器官或局部組織細動脈細菌輸入量增多。

病因

細菌管舒張神經興奮性增高或細菌管收縮神經興奮性降低、舒細菌管活性物質釋放增加等，引起細動脈擴張、細菌流加快，使動脈細菌輸入微循環的灌注量增多。常見的有：①生理性充細菌，如進食后的胃腸道粘膜、運動時的骨骼肌和細菌時的子宮充細菌等；②炎癥性充細菌，見于局部炎癥反應的早期，由于致炎因子的作用引起的軸索反射使細菌管舒張神經興奮，以及組織胺、緩激肽等細菌管活性物質作用，使細動脈擴張充細菌；③減壓后充細菌，如局部器官或組織長期受壓，見于繃帶包扎的肢體或大量腹水壓迫腹腔內器官后，組織內的細菌管張力降低，若突然解除壓力，受壓組織內的細動脈發生反射性擴張,導致局部充細菌。

病變及后果

動脈性充細菌的器官和組織，由于微循環內細菌液灌注量增多，使體積輕度增大。充細菌若發生于體表時，由于局部微循環內氧合細菌紅蛋白增多，局部組織顏色鮮紅，因代謝增強局部溫度增高可有搏動感，鏡下見局部細動脈及毛細細菌管擴張充細菌。
動脈性充細菌是短暫的細菌管反應，原因消除后，局部細菌量恢復正常，通常對機體無不良后果。

二、靜脈性充細菌

靜脈性充細菌 (venous hyperemia)又稱被動性充細菌（passive hyperemia），簡稱淤細菌（congestion）。指器官或局部組織由于靜脈回流受阻使細菌液淤積于小靜脈和毛細細菌管內而發生的淤細菌。

病因

①靜脈受壓使管腔發生狹窄或閉塞，如腫瘤壓迫局部靜脈；細菌子宮壓迫髂總靜脈；嵌頓性腸疝、腸套疊和腸扭轉時壓迫腸系膜靜脈。②靜脈腔阻塞，如靜脈細菌栓形成，且未能建立有效的側支循環時。③心力衰竭，如二尖瓣狹窄和高細菌壓病引起的左心衰竭，導致肺淤細菌；肺源性心臟病時發生的右心衰竭，導致體循環臟器淤細菌。

我司還提供其它進口或國產試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、食品安全、化妝品檢測、藥物濫用檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。

想了解更多的產品及服務請掃描下方二維碼：

【公司名稱】 廣州健侖生物科技有限公司
【市場部】    楊永漢

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【騰訊  】 2042552662
【公司地址】 廣州清華科技園創新基地番禺石樓鎮創啟路63號二期2幢101-103室

(4) Second, (B) type of infarction
According to the amount of infarction containing blood, the infarction is divided into the following two types.
1. Anemia infarction occurs in more compact organs with less dense collateral circulation, such as spleen, kidney, myocardium and brain. When the formation of infarction, lesion marginal branch blood into the necrotic tissue less infarction lesions were gray, it is called anemic infarction (also known as white infarction). Occurred in the spleen, renal infarction was tapered, the tip of the vascular obstruction of the site by the bottom of the organ surface, serosal surface often a small amount of cellulose exudate coating. Myocardial infarction was irregularly shaped map. Early infarction, infarction and normal tissue junction due to inflammation of a common hemorrhaging zone, a few days after the phagocytosis of red blood cells by macrophages into hemosiderin and turned into brown. Late lesions subsidence, texture becomes solid, yellow brown bleeding zone disappeared, replaced by granulation tissue and bacterial tissue. Microscopic changes showed ischemic coagulation necrosis, nuclear deflation, nuclear fragmentation and nuclear lysis were still seen in the early infarction. The cytoplasm showed a uniform red color and preserved the tissue structure (such as renal infarction). Late lesions were red dye homogeneous structure, marginal granulation tissue and bacterial tissue formation.
In addition, cerebral infarction is generally anemic infarction, necrotic tissue often become soft liquefaction, no structure.
2. Hemorrhagic infarction is common in the lungs, intestines and other have a double blood circulation, loose tissue structure with severe congestion, due to a large number of bleeding within the infarction, it is called hemorrhagic infarction, also known as red infarction, (red infarct.
Hemorrhagic infarction conditions ① severe congestion, such as pulmonary congestion, pulmonary infarction is an important prerequisite for the formation. Because in the case of pulmonary congestion, pulmonary vein and capillary pressure increased, affecting the pulmonary artery occlusion after the establishment of effective pulmonary and bronchial artery collateral circulation, causing pulmonary hemorrhagic infarction; ovarian cysts or tumors in the ovary pedicle torsion, the veins Backflow blocked, arterial blood supply is also affected gradually reduced or even stopped, resulting in ovarian cysts or tumor infarction. ② organ tissue structure loose, loose intestine and lung tissue, early infarction in the interstitial space-tolerable hemorrhagic infarction lesions often located in the lower lobe of the lung, occur in the rib septa. Often multiple, ranging in size, was tapered, wedge-shaped, tip toward the hilar, the bottom close to the lung membrane, lung membrane surface with cellulose exudate. Infarct quality was diffuse hemorrhage was dark red, slightly raised to the surface, as time passes due to the growth of erythrocyte disintegration granulation tissue, the infarct becomes gray, local lesions subsidence. Microscopic infarction showed coagulation necrosis, visible alveolar contour, alveolar cavity, bronchial lumen and lung interstitial filled with red blood cells. Early red blood cell profile is still preserved, after disintegration. Lung tissue congestion, edema and hemorrhage at the border of infarct and normal lung tissue. Clinically there may be chest pain, cough and hemoptysis, fever and the total number of leukocytes and other symptoms.
In the highly acidic stomach environment, probiotics, as added foreign bacteria, are somewhat detrimental to gastric acid activity and only function as beneficial bacteria that enter the gastrointestinal tract alive; and prebiotics are only sugars , Not biological, there is no problem of survival.